Τοποθεσία - Ψαρού

Αποστάσεις

Τα 'Studios Ψαρού' βρίσκονται μπροστά από την παραλία της Ψαρού
(πλούσια σε άργυλο) στην βορειοανατολική ακτή του νησιού, ανάμεσα
από το Τσιλιβί και το Αμμούδι και 13 χλ από την πόλη της Ζακύνθου.
Το χωριό της Ψαρού και η γύρω περιοχή είναι πολύ ήσυχη,
καταπράσινη και προσφέρεται για περιπάτους. Σε απόσταση 200
μέτρων από τα δωμάτια υπάρχουν παραδοσιακές ταβέρνες και μίνι
μάρκετ. Στα 400 μέτρα υπάρχει ένα ήσυχο παραθαλάσσιο μπαράκι.
Γραφεία ενοικιάσεων αυτοκινήτων, ποδηλάτων και μηχανών
βρίσκονται στα 600 μέτρα. Επιπλέον κοντά στα δωμάτια υπάρχει το
λιμανάκι 'Κάβος' όπου μπορείτε να δέσετε τη βάρκα ή το σκάφος σας
δωρεάν. Τα 'Studios Ψαρού' απέχουν 1 χλμ. από τα 'Studios
Γερακάρι'.
•  Παραλία : 0 μ
•  Εστιατόριο : 200 μ
•  Μίνι μάρκετ: 200 μ
•  Beach Bar : 400 μ
•  Πισίνα : 300 μ
•  Στάση λεωφορείου: 200 μ
•  Λιμανάκι 'Κάβος' : 500 μ
•  Πόλη και λιμάνι Ζακύνθου: 13 xλμ
•  Αεροδρόμιο (Λαγανάς): 15 xλμ
•  Νοσοκομείο Ζακύνθου: 13 χλμ
•  Κοντινότερο φαρμακείο: Τσιλιβί (4 χλμ)
•  Κοντινότερο ΑΤΜ: Τσιλιβί (4 χλμ)

Δραστηριότητες

•  Tennis club: Τραγάκι (3 χλμ)
•  Ιππασία: Τραγάκι (3 χλμ)
•  Θαλάσσια σπορ: Τσιλιβί (4 χλμ) και Αλυκές (3 χλμ)
•  Νεροτσουλήθρες: Τσιλιβί (4 χλμ) και Σαρακηνάδο (12 χλμ)
•  Bowling : Τσιλιβί (4 χλμ)
•  Τουριστική αγορά: Τσιλιβί (4 χλμ)
•  Νυχτερινή διασκέδαση: Τσιλιβί (4 χλμ) και Αλυκές (3 χλμ)

Χάρτης

Στο χάρτη μπορείτε να διακρίνετε τη διαδρομή από το αεροδρόμιο και το λιμάνι της Ζακύνθου προς τα δωμάτιά μας στην Ψαρού
(πράσινο σύμβολο) και στο Γερακάρι (κόκκινο σύμβολο). Η διαδρομή που θα ακολουθήσετε είναι με τη σειρά: αεροδρόμιο, λιμάνι
Ζακυνθου, Ακρωτήρι, Τσιλιβί (Πλάνος), Τραγάκι (το παραλιακό τμήμα του χωριού), Κυψέλη (το παραλιακό τμήμα του χωριού),
Γερακάρι και Ψαρού.

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STUDIOS ΨΑΡΟΥ
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Τρίκλινα
Τοποθεσία
Οικολογία - Εθνικό Θαλάσσιο Πάρκο Ζακύνθου
Νομαρχιακή αυτοδιοίκηση Ζακύνθου
Αεροπορικά δρομολόγια - Oλυμπιακή
ΚΤΕΛ νομού Ζακύνθου – Yπεραστικά δρομολόγια
Πλοία προς την Ζάκυνθο - τιμές και δρομολόγια

Χρήσιμοι Σύνδεσμοι (διαδικτυακοί τόποι)

Μέσο Γερακάρι - 29100 Ζάκυνθος, Ιόνια Νησιά T: +30 26950 63023 (καλοκαίρι) T: +30 26950 44542 (χειμώνας) E: thalassa.studios@yahoo.gr

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Olanzapine 10 mg sleep medication. However, the patient refused to take medication. A sleep analysis, including an electroencephalogram was performed and revealed profound alterations in circadian and paracrine behavior. This patient also experienced hallucinations and nightmares after discontinuation of medication. Therefore, the patient's response to psychotherapy was evaluated, and treatment initiated to establish a more adequate treatment regimen. In this case, a reduction drug dosage was advised to obtain a response. The drug was reduced from 10 mg to 5 daily, gradually reducing the maximum olanzapine brand names australia dose to 5 mg/day. At baseline a patient had been using approximately 30 mg per night of Zolpidem, a dose that was initially established at a low level of anxiety. The treatment was continued daily in an effort to get a lower dosage level. The drug doses were reduced and at the end of first week patient was off all pharmacological treatments and showed a dramatic improvement in his symptoms. The patient was discharged from hospital with an effective treatment for a sleep disorder. The present case represents one of the few reported cases sleep-related psychosis in which the patient refused to participate in psychotherapy. other cases, patients refused to participate in therapy and were treated with antidepressants. A case of insomnia with possible sleep-related psychosomatic basis in a patient with bipolar I disorder was described [13]. No other report from our clinic describes a patient whose refusal to undergo psychotherapeutic treatment was a cause for termination of sleep-related psychotherapy. The mechanism of sleep-related psychosis in schizophrenia is Olanzapine 2.5mg $67.03 - $0.74 Per pill not well understood. One hypothesis is that a psychomotor response to sleep deprivation might play a role, thereby explaining the occurrence of Tranexamic acid to buy uk sleep-related psychoses as well the failure to benefit from pharmacological treatment [26]. Sleep deprivation can lead to increased levels of prolactin and decreased brain concentrations of dopamine, serotonin, noradrenalin, and melatonin. Dopaminergic, serotonergic, melatoninergic mechanisms may be affected, resulting in a negative sleep-related psychosomatic syndrome. Sleep deprivation may also provoke an acute psychotic reaction, which may be misinterpreted as sleep-related psychosis (see below). Several lines of evidence strongly suggest that sleep-related disorders can constitute a part of bipolar I illness. In addition to the clinical data, it has been suggested that there are genetic predisposition factors for sleep-related disorders [27], [28]. example, the polycystic ovaries syndrome has been linked to a high incidence of sleep related disorders in women with bipolar I or II disorder [27]. Additionally, the co-occurrence of other psychiatric disorders (mood stabilizers, sedatives, and anticonvulsants Online viagra germany or atypical antipsychotics) sleep-related disorders has been noted in patients with bipolar I disorder [29]. Furthermore, it has been reported that schizophrenia patients olanzapine in australia who have slept in hospital the last four months have increased risk.



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